Pathology illustrated

abnormal acid activity acute alveoli anaemia antibodies antigen appearance arise arteries associated atrophy bacteria become blood body bone capillaries carcinoma cardiac cause cells changes chromosomes chronic clinical common complex complications condition containing continued cytoplasm damage def...

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Bibliographic Details
Main Authors: Govan, Alasdair D. T. (Author), Callander, Robin (Author), Macfarlane, Peter S. (Author)
Format: Book
Published: Edinburgh, London Churchill Livingstone 1986
Edition:Second Edition
Subjects:
Online Access:Click Here to View Status and Holdings.
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245 1 0 |a Pathology illustrated  |c Alasdair D. T. Govan, Peter S. Macfarlane, Robin Callander 
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520 # # |a abnormal acid activity acute alveoli anaemia antibodies antigen appearance arise arteries associated atrophy bacteria become blood body bone capillaries carcinoma cardiac cause cells changes chromosomes chronic clinical common complex complications condition containing continued cytoplasm damage deficiency degeneration deposition dilatation disease disorders duct early effects enlargement enzymes epithelium especially essential example excessive factors failure fibres fibrin fibrosis fibrous fluid formation function genes glands glucose groups growth haemorrhage healing heart hepatic immune important increased infarction infection inflammation inflammatory intestine involved kidney leading lesions liver loss lung lymphocytes macrophages malignant marrow mechanisms membrane muscle necrosis nerve neutrophils nodes normal Note obstruction occasionally occurs organs particularly pathological pigment plasma present pressure primary production progressive proliferation protein rare reaction red cells renal response result secondary secretion seen severe skin specific spread stage surface tissue tract tumour ulcer usually vessels virus wall 
650 # 0 |a Pathology 
700 1 # |a Callander, Robin  |e author 
700 1 # |a Macfarlane, Peter S.  |e author 
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